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Reducing synuclein accumulation improves neuronal survival after spinal cord injury

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dc.contributor.author Fogerson, Stephanie M.
dc.contributor.author van Brummen, Alexandra J.
dc.contributor.author Busch, David J.
dc.contributor.author Allen, Scott R.
dc.contributor.author Roychaudhuri, Robin
dc.contributor.author Banks, Susan M. L.
dc.contributor.author Klärner, Frank-Gerrit
dc.contributor.author Schrader, Thomas
dc.contributor.author Bitan, Gal
dc.contributor.author Morgan, Jennifer R.
dc.date.accessioned 2018-03-28T00:07:52Z
dc.date.available 2018-03-28T00:07:52Z
dc.date.issued 2016-04
dc.identifier.citation Fogerson, S. M., van Brummen, A. J., Busch, D. J., Allen, S. R., Roychaudhuri, R., Banks, S. M., & ... Morgan, J. R. (2016). Reducing synuclein accumulation improves neuronal survival after spinal cord injury. Experimental Neurology, 278105-115. doi:10.1016/j.expneurol.2016.02.004 en_US
dc.identifier.issn 0014-4886
dc.identifier.uri http://hdl.handle.net/11416/376
dc.identifier.uri https://search.ebscohost.com/login.aspx?direct=true&AuthType=shib&db=edselp&AN=S0014488616300267&site=eds-live&scope=site&custid=s5615486
dc.description.abstract Spinal cord injury causes neuronal death, limiting subsequent regeneration and recovery. Thus, there is a need to develop strategies for improving neuronal survival after injury. Relative to our understanding of axon regeneration, comparatively little is known about the mechanisms that promote the survival of damaged neurons. To address this, we took advantage of lamprey giant reticulospinal neurons whose large size permits detailed examination of post-injury molecular responses at the level of individual, identified cells.[...] en_US
dc.language.iso en_US en_US
dc.publisher Elsevier Inc. en_US
dc.subject CLR01 en_US
dc.subject Lamprey en_US
dc.subject Molecular tweezer en_US
dc.subject Neurodegeneration en_US
dc.subject Parkinson's disease en_US
dc.subject Synaptotagmin en_US
dc.title Reducing synuclein accumulation improves neuronal survival after spinal cord injury en_US
dc.type Article en_US

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